Theory of macrophage injury: Lung p

Theory of macrophage injury: Lung phagocytes are distributed widely on the surfaces of bronchial alveoli, which can form a barrier against the external environment so as to reach its aim of protecting human body via swallowing the inhalable PM and pathogenic microorganisms. Meanwhile, they are one of the critical factors in maintaining the aseptic environment in lung because they can participate in the immunoregulatory process through playing the antigen presenting effect so as to activate the innate immunity. Lundborg et al (Lundborg et al., 2006) discovered that with the increase of carbon black PM dosage, the swallowing and adsorbing abilities of phagocytes decreased markedly in human body, indicating that PM could decrease the protective function of phagocytes by reducing their swallowing ability.
Difference analysis of SO2 concentrations on the number of visiting patients in Department of Respiratory
SO2, as one of the important air pollutants, is correlated with the gas emissions of motor vehicles. As a water-soluble stimulating gas, SO2 can be dissolved into the watery mucus of upper respiratory tract and bronchial mucous, which then is turned into sulfite or bisulfate and absorbed into blood. Then, it will rapidly spread to the whole body, stimulate the smooth muscle of respiratory tract, contract trachea and bronchus and increase the airway resistance and secretions. People who are exposed in high-concentration SO2 will have symptoms of wheeze and short breath, etc.

結果 (中文) 1:

巨噬细胞损伤理论: 肺巨噬细胞在支气管肺泡，可以形成一个外部的环境，以达到其目的是保护人体通过吞咽可吸入 PM 的屏障的表面分布广泛和致病性微生物。同时，它们是维持肺的无菌环境，因为他们可以参与免疫调节过程通过玩抗原呈递，激活先天免疫效果的关键因素之一。Lundborg et al (Lundborg et al，2006年) 发现，随着炭黑 PM 剂量、 吞咽和吸附能力明显下降，在人体内的吞噬细胞的增加表明 PM 可以通过减少他们吞咽能力降低巨噬细胞的保护作用。SO2 浓度对探望病人在呼吸科的数目的差异分析作为一个重要的空气中的污染物，SO2，被相关的气体排放量机动车辆。作为一种水溶性的刺激气体，SO2 能溶解成水汪汪粘液的上呼吸道和支气管黏膜，然后是变成亚硫酸或硫酸氢钠和吸收，进入血液。然后，它将迅速蔓延到全身，刺激呼吸道、 合同气管和支气管平滑肌和增加气道阻力和分泌物。暴露在高浓度 SO2 的人会有喘息、 气促等症状。

結果 (中文) 3:

巨噬细胞损伤理论：肺巨噬细胞对支气管肺泡表面分布广泛，可形成对外部环境的屏障，从而达到保护人体的目的通过吞咽的可吸入颗粒物和病原微生物。同时，他们在维持肺的无菌环境的关键因素之一，因为他们可以参与免疫调节过程在抗原提呈作用从而激活先天免疫。Lundborg等人（Lundborg et al.，2006）发现，随着炭黑用量的增加，吞咽时，吸附在人体细胞的能力明显降低，表明PM可以通过减少吞咽能力降低吞噬细胞的保护作用。二氧化硫浓度对呼吸科就诊患者数量的差异分析作为一种重要的大气污染物之一，其与机动车尾气排放的关系密切相关。作为一种水溶性的刺激性气体，SO2可溶解于上呼吸道和支气管粘液稀粘液，然后转化为亚硫酸盐或亚硫酸氢盐和吸收到血液。然后，它会迅速扩散到全身，刺激呼吸道的平滑肌，气管和支气管收缩，增加气道阻力和分泌物。人在高浓度SO2暴露会有喘息、气促等症状，