AMPH administration resulted in a m

AMPH administration resulted in a marked hyperactivity and increased oxidative stress, as manifested by increased SOD activity, decreased activities of CAT and GPx, reduced levels of NPSH and increased levels of TBARS and PC. The administration of anti-ouabain antibodies, which reduced the AMPH-induced hyperactivity, protected against the concomitant oxidative stress in the brain.

結果 (繁體中文) 1:

AMPH給藥導致的明顯多動和氧化應激增加，通過增加的超氧化物歧化酶活性的表現，降低，CAT和GPX的活動，減少NPSH水平增加TBARS和PC的水平。抗哇巴因抗體的管理，減少了苯丙胺誘導多動，對在大腦中伴隨氧化應激的保護。

結果 (繁體中文) 2:

AMPH管理導致明顯的多動和氧化應激增加，表現為SOD活性增加，CAT和GPx活動減少，NPSH水準降低，TBARS和PC水準增加。抗歐巴因抗體的產生，減少了AMPH引起的多動性，防止了大腦中伴隨的氧化應激。

結果 (繁體中文) 3:

注射苯丙胺導致明顯的多動症和氧化應激新增，表現為SOD活性新增，CAT和GPx活性降低，NPH水准降低，TBAR和PC水准升高。注射抗哇巴因抗體，降低苯丙胺誘發的多動症，保護大腦免受伴隨的氧化應激。