As shown in Figure 5, the basal aor

As shown in Figure 5, the basal aortic superoxide anion production was significantly increased in either glucose-fed rats (P < 0.01) or glucose-fed rats also treated with corn oil (P < 0.05). Oral treatment with argan oil prevented this increase in glucose-fed rats, so that the basal superoxide anion production did not significantly differ from that in control rats (Fig. 5). As shown in Figure 6, the 5-wk glucose feeding, with or without corn oil, significantly elevated (P < 0.01) NADPH oxidase activity in aorta tissue. Chronic treatment with argan oil prevented an increase in NADPH oxidase activity in glucose-fed rats so that the aortic NADPH oxidase activity did not significantly differ from that in control rats (Fig. 6). Treatment with DPI, a selective inhibitor of NADPH oxidase, blunted all changes in the activity of the aortic NADPH oxidase in the four groups of rats (Fig. 6).

結果 (繁體中文) 1:

如圖 5 所示，產生基部主動脈超氧陰離子顯著增高或葡萄糖喂大鼠 (P < 0.01) 或葡萄糖飼養大鼠也用玉米油治療 (P < 0.05)。摩洛哥堅果油口服治療阻止葡萄糖餵食的老鼠，這種增加，產生基超氧陰離子沒有明顯的差別，在對照組大鼠 (圖 5)。5 wk 葡萄糖餵養，有或沒有玉米油，圖 6 中所示明顯升高 (P < 0.01) 主動脈組織中的 NADPH 氧化酶活性。摩洛哥堅果油慢性治療防止增加葡萄糖喂大鼠 NADPH 氧化酶活性，主動脈 NADPH 氧化酶活性沒有明顯的差別，在對照組大鼠 (圖 6)。DPI，NADPH 氧化酶，選擇性抑制劑治療鈍所有四組大鼠 (圖 6) 主動脈的 NADPH 氧化酶活性的變化。

結果 (繁體中文) 2:

如圖5所示，基底主動脈產生超氧陰離子在任葡萄糖餵養大鼠（P <0.01）或葡萄糖餵養大鼠也與玉米油（P <0.05）處理過顯著增加。與堅果油口服治療防止這種增加葡萄糖餵養大鼠，使得基底產生超氧陰離子沒有顯著不同於在對照大鼠（圖5）不同。主動脈組織中如在圖6中，5週葡萄糖進料，具有或不具有玉米油中所示，顯著升高（P <0.01），NADPH氧化酶活性。與堅果油慢性治療防止葡萄糖餵養大鼠的增加NADPH氧化酶活性，使得主動脈NADPH氧化酶活性沒有顯著不同於在對照大鼠差異（圖6）。與新聞部，NADPH氧化酶的選擇性抑製劑，治療減弱在主動脈NADPH氧化酶的活性在四組大鼠（圖6）的所有的變化。